By G. Ying
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Additional resources for Age-Related Macular Degeneration - Recent Advs. in Basic Research, Clin. Care
2007) first promote activation of src-family kinases that trigger NF-κB- and AP-1-dependent transcription via small G-protein-MAPK pathways. Although the mechanistic details of RAGE signaling and the importance of its various ligands in disease pathology continue to be areas of investigation, activated RAGE by its various ligands also leads to activation of transcription factors NF-κB and AP-1 by ras-MAPK signaling (Glenn & Stitt, 2009). Activated NF-κB and AP-1 finally drive expression of inflammatory mediators, promoting sterile inflammation.
2006 May;90(5):593-6. A. (1989). Analysis of newly synthesized Bruch's membrane proteoglycans. Invest Ophthalmol Vis Sci 30,478-486. V. (1997). Reattachment of cultured human retinal pigment epithelium to extracellular matrix and human Bruch's membrane. Invest Ophthalmol Vis Sci 38, 1110-1118. Hua J, Spee C, Kase S, Rennel ES, Magnussen AL, Qiu Y, Varey A, Dhayade S, Churchill AJ, Harper SJ, Bates DO, Hinton DR. Recombinant human VEGF165b inhibits experimental choroidal neovascularization. Invest Ophthalmol Vis Sci.
However, whether there is a common mechanism by which numerous heterogeneous stimuli converge on NLRP3 downstream of ROS generation, potassium efflux and lysosomal rupture, remains to be determined. 3 Production of active IL-1β by AIM2 inflammasome In addition to the NLRP3 inflammasome, the AIM2 inflammasome is the second one indentified so far to be involved in sterile inflammation. In contrast to NLRP3, AIM2 has a highly restricted spectrum of activating stimuli, currently known being involved in sensing cytosolic dsDNA regardless of the DNA source.
Age-Related Macular Degeneration - Recent Advs. in Basic Research, Clin. Care by G. Ying